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most people who break a bone make a full recovery and can resume their typical routine after their bone heals. some fractures can have a long-term impact on your life, especially if you experienced other injuries. talk to your surgeon or provider before resuming any physical activities or playing sports while youre recovering.
for athletes, the goals of rehabilitation are to return to your sport with the greatest chance of a successful outcome. this is the time to start your strength training. do not attempt to return to your sport before the injured muscle is fully healed, and do not attempt to return to your sport before you have full range of motion and strength.
the first step is prevention. to prevent injuries, watch for places where you are over-reaching, be careful of over-training, and make sure that your nutrition and hydration are in line with your goals. proper nutrition and hydration allow you to train harder, recover faster, and allow you to train in longer sessions. your sports nutrition and hydration program should include: pre-, peri- and post-exercise nutrition and hydration are important to athletes and should be planned carefully.
if your fracture is mild and your bones did not move far out of place (if its non-displaced), you might only need a splint or cast. splinting usually lasts for three to five weeks. if you need a cast, it will likely be for longer, typically six to eight weeks. in both cases youll likely need follow up x-rays to make sure your bones are healing correctly.
return to full activity is usually allowed when the patient is pain free, has full range of motion, and full strength. if an athlete attempts to return to their sport before these criteria are met, there is a high chance of reinjuring the muscle and sustaining a setback. mild, grade 1 injuries may require only two to three weeks before an athlete can return. more severe injuries may require significantly more time.
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Thymosin β4 (Tβ4) is secreted by platelets and plays an important role in wound healing, and an increased platelet count is a characteristic of tumor-induced thrombocytosis. However, the role of platelets in tumor progression is unclear. In the present study, we focused on the relationship between platelets and tumor progression using a patient with advanced non-small cell lung cancer (NSCLC). For the patient, the thymosin-β4 level was evaluated by enzyme-linked immunosorbent assay (ELISA) before and after the first cycle of chemotherapy. Although the patient was free of systemic inflammatory response at the time of admission, the plasma Tβ4 level was high, suggesting the presence of tumor-derived Tβ4. The platelet count in the patient was consistently high at various time points before, during, and after chemotherapy, suggesting tumor-induced thrombocytosis. Some patients with various solid tumors have been reported to show elevated levels of Tβ4 in their blood. However, the present patient showed the highest level in his blood at the time of diagnosis. Although no thymosin-β4-producing tumor cells were detected by immunohistochemistry, we can postulate that Tβ4 was secreted from the platelets of the tumor cells and that the former may help activate the tumor cells. Increased platelet counts were observed in the same patient during the
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